Proteus BioSciences 25-6790 - Myosin-VIIa 现货供应-试剂-金山科研平台

Proteus BioSciences 25-6790 - Myosin-VIIa 现货供应

2025-08-01

Proteus BioSciences 25-6790 - Myosin-VIIa 现货供应

金山科研平台代理微信：jinshanbioProteus BioSciences was founded in 2002 to develop innovative products to advance proteomic research. We specialize in discovering peptides and proteins with novel biological properties

Myosin-VIIaRabbit Polyclonal Antibody

PRODUCT #: 25-6790

DESCRIPTION: Myosin-VIIa is one of the unconventional members of the myosin molecular motor superfamily that move along filamentous actin. The protein is expressed primarily in the inner ear, retina and testis. Defects in the myosin-VIIa gene （MYO7A） are responsible for Usher syndrome, characterized by hearing impairment, lack of vestibular function and progressive retinal degeneration.MYO7A mutations are are responsible for deafness in shaker-1 mice and in other model systems. In the inner ear, myosin-VIIa is required for assembly of stereocilia, the sensory structures necessary for detecting sound signals.
SPECIES CROSS-REACTIVITY:	Human, mouse, rat, pig, avian, amphibian
APPLICATIONS/DILUTIONS:	IF （5-10 ug/ml） WB （0.5-1.0 ug/ml）
SOURCE:	Rabbits were immunized with amino acids 880-1077 from the tail region of human myosin-VIIa.
FORM/STORAGE:	50 ug （0.5 mg/ml） of affinity purified IgG with 50% glycerol, 0.01% sodium azide and 1.0 mg/ml BSA. Store at -20º C. Avoid multiple freeze/thaw cycles.
References: Hasson, T. et al. （1995）, Proc. Natl. Acad. Sci. 92:9815-9819. Hasson, T. et al. （1997）, Journal of Cell Biology 137:1287-1307. Hasson, T. et al. （1997）, Cell Motil. Cytoskel. 37:127-138. Hasson, T. et al. （2005）, Cell Motil. Cytoskel. 62:13-26.

DESCRIPTION: Myosin-VIIa is one of the unconventional members of the myosin molecular motor superfamily that move along filamentous actin. The protein is expressed primarily in the inner ear, retina and testis. Defects in the myosin-VIIa gene （MYO7A） are responsible for Usher syndrome, characterized by hearing impairment, lack of vestibular function and progressive retinal degeneration.MYO7A mutations are are responsible for deafness in shaker-1 mice and in other model systems. In the inner ear, myosin-VIIa is required for assembly of stereocilia, the sensory structures necessary for detecting sound signals.
SPECIES CROSS-REACTIVITY:	Human, mouse, rat, pig, avian, amphibian
APPLICATIONS/DILUTIONS:	IF （5-10 ug/ml） WB （0.5-1.0 ug/ml）
SOURCE:	Rabbits were immunized with amino acids 880-1077 from the tail region of human myosin-VIIa.
FORM/STORAGE:	50 ug （0.5 mg/ml） of affinity purified IgG with 50% glycerol, 0.01% sodium azide and 1.0 mg/ml BSA. Store at -20º C. Avoid multiple freeze/thaw cycles.
References: Hasson, T. et al. （1995）, Proc. Natl. Acad. Sci. 92:9815-9819. Hasson, T. et al. （1997）, Journal of Cell Biology 137:1287-1307. Hasson, T. et al. （1997）, Cell Motil. Cytoskel. 37:127-138. Hasson, T. et al. （2005）, Cell Motil. Cytoskel. 62:13-26.

DESCRIPTION: Myosin-VIIa is one of the unconventional members of the myosin molecular motor superfamily that move along filamentous actin. The protein is expressed primarily in the inner ear, retina and testis. Defects in the myosin-VIIa gene （MYO7A） are responsible for Usher syndrome, characterized by hearing impairment, lack of vestibular function and progressive retinal degeneration.MYO7A mutations are are responsible for deafness in shaker-1 mice and in other model systems. In the inner ear, myosin-VIIa is required for assembly of stereocilia, the sensory structures necessary for detecting sound signals.
SPECIES CROSS-REACTIVITY:	Human, mouse, rat, pig, avian, amphibian
APPLICATIONS/DILUTIONS:	IF （5-10 ug/ml） WB （0.5-1.0 ug/ml）
SOURCE:	Rabbits were immunized with amino acids 880-1077 from the tail region of human myosin-VIIa.
FORM/STORAGE:	50 ug （0.5 mg/ml） of affinity purified IgG with 50% glycerol, 0.01% sodium azide and 1.0 mg/ml BSA. Store at -20º C. Avoid multiple freeze/thaw cycles.
References: Hasson, T. et al. （1995）, Proc. Natl. Acad. Sci. 92:9815-9819. Hasson, T. et al. （1997）, Journal of Cell Biology 137:1287-1307. Hasson, T. et al. （1997）, Cell Motil. Cytoskel. 37:127-138. Hasson, T. et al. （2005）, Cell Motil. Cytoskel. 62:13-26.

DESCRIPTION: Myosin-VIIa is one of the unconventional members of the myosin molecular motor superfamily that move along filamentous actin. The protein is expressed primarily in the inner ear, retina and testis. Defects in the myosin-VIIa gene （MYO7A） are responsible for Usher syndrome, characterized by hearing impairment, lack of vestibular function and progressive retinal degeneration.MYO7A mutations are are responsible for deafness in shaker-1 mice and in other model systems. In the inner ear, myosin-VIIa is required for assembly of stereocilia, the sensory structures necessary for detecting sound signals.
SPECIES CROSS-REACTIVITY:	Human, mouse, rat, pig, avian, amphibian
APPLICATIONS/DILUTIONS:	IF （5-10 ug/ml） WB （0.5-1.0 ug/ml）
SOURCE:	Rabbits were immunized with amino acids 880-1077 from the tail region of human myosin-VIIa.
FORM/STORAGE:	50 ug （0.5 mg/ml） of affinity purified IgG with 50% glycerol, 0.01% sodium azide and 1.0 mg/ml BSA. Store at -20º C. Avoid multiple freeze/thaw cycles.
References: Hasson, T. et al. （1995）, Proc. Natl. Acad. Sci. 92:9815-9819. Hasson, T. et al. （1997）, Journal of Cell Biology 137:1287-1307. Hasson, T. et al. （1997）, Cell Motil. Cytoskel. 37:127-138. Hasson, T. et al. （2005）, Cell Motil. Cytoskel. 62:13-26.

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